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development of IAA was regarded as possible on account of oxidase deamination of NMT (Barker et al., 1980), but was later on established to become also partly by direct deamination by MAO (Barker et al., 1982). Pretreatment with the MAO-I iproniazid in rat full brain homogenates inhibited IAA development by eighty three%, NMT and DMT-NO formation w
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